Why is "11β-hydroxylase deficiency" trending?
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Trend Analysis
- Ranking position: #
- Date: 2026-04-02 07:16:57
This topic has appeared in the trending rankings 1 time(s) in the past year. While it does not trend frequently, its appearance suggests a renewed or concentrated surge of public interest.
Based on Wikipedia pageviews and search interest, this topic gained significant attention on the selected date.
Trend Insight
This topic is not currently in the ranking.
Wikipedia Overview
Congenital adrenal hyperplasia due to 11β-hydroxylase deficiency is a form of congenital adrenal hyperplasia (CAH) which produces a higher than normal amount of androgen, resulting from a defect in the gene encoding the enzyme steroid 11β-hydroxylase (11β-OH) which mediates the final step of cortisol synthesis in the adrenal. 11β-OH CAH results in hypertension due to excessive mineralocorticoid effects. It also causes excessive androgen production both before and after birth and can virilize a genetically female fetus or a child of either sex.
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Search Interest Perspective
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Congenital adrenal hyperplasia due to 11-beta-hydroxylase deficiency: clinical, biochemical and molecular characteristics and long-term outcomes - analesdepediatria.org
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The Complex Spectrum of 11β-Hydroxylase Deficiency: A Case of Precocious Puberty, Hypertension, and Testicular Adrenal Rest Tumors (TARTs) - Cureus
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Clinical and Molecular Analysis of Four Patients With 11β-Hydroxylase Deficiency - Frontiers
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Acquired 11β-hydroxylase Deficiency by Inhaled Etomidate and its Analogues - Endocrine Society
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Congenital adrenal hyperplasia due to 11-beta-hydroxylase deficiency: functional consequences of four CYP11B1 mutations - Nature
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Adrenocortical 11β-Hydroxylase Deficiency and Virilism First Manifest in the Adult Woman - The New England Journal of Medicine
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From Severe Virilization to Precocious Puberty in a 46,XX Patient: Therapeutic Challenges in a Case of 11β-Hydroxylase Deficiency - Cureus
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A Novel Mutation in the CYP11B1 Gene Causes Steroid 11β‐Hydroxylase Deficient Congenital Adrenal Hyperplasia with Reversible Cardiomyopathy - Wiley Online Library
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Figure 8. [46,XX DSD decision tree diagram.]. - Endotext - ncbi.nlm.nih.gov
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Clinical and molecular characterization of 10 Chinese children with congenital adrenal hyperplasia due to 11beta-hydroxylase deficiency - springermedizin.de
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Classical 11β-Hydroxylase Deficiency Caused by a Novel Homozygous Mutation: A Case Study and Literature Review - Cureus
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Challenges in the treatment of late-identified untreated congenital adrenal hyperplasia due to CYP11B1 deficiency: Lessons from a developing country - Frontiers
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Secondary Biosynthetic Defects in Women with Late-Onset Congenital Adrenal Hyperplasia - The New England Journal of Medicine
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Male Gender Identity and Reversible Hypokalemic Hypertension in a 46,XX Child with 11-Beta-Hydroxylase Deficiency Congenital Adrenal Hyperplasia - Cureus
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